Medical Attributes of Ephedra sp. – Ephedra / Ma-huang

by Cassandra Cragle
Wilkes University
Wilkes-Barre, PA

July 2015


Ephedra spp., commonly known as Chinese ephedra or ma-huang, are shrubby species of the genus Ephedra L. within the Ephedraceae (ephedra family) (Bell & Bachman 2011).  It is native to Central Asia, including southern Siberia (Russian Federation), Mongolia, and China (Bell & Bachman 2011).  Ephedra can also be found in the southwestern United States, temperate tropical regions of North America and Mediterranean regions, Mexico, South America (Ecuador to Patagonia and Argentina), southern Europe, and northern Africa (Bell & Bachman 2011). Other common names for Ephedra include Mormon-tea, jointfir, cañatilla, popotillo, tepopote, as well as many others (FNA n.d., ITIS 2010).

While typically a shrub, Ephedra is occasionally a clambering vine, which has jointed branches that are yellowish-green to olive-green when young. The leaves are opposite or in whorls of 3 (FNA n.d.). It is terrestrial, found in arid areas and highlands, on slopes, dry riverbeds, in sandy locations, or in fields on mountainsides. Classified as a gymnosperm (Order Gnetales), Ephedra produces cones and wind pollinates from March to June and sets seed between June through September (Bell & Bachman 2011). The plants are dioecious, female and male cones being produced on different plants, and are wind-pollinated. All plants in a population tend to reproduce in synchronization during a year with adequate rainfall, and then not again for several years (Meyer 2015).

As an herbal remedy, the stems have shown anti-inflammatory activity. It also possesses antitussive effects aiding in coughing, cardiovascular effects and genetic effects that can mutate about 100 genes (Sigma-Aldrich 2015). Ephedra sinica extracts also possess outstanding anti-bacterial activity (Dashtdar, et al. 2013). It is also used to treat asthma, nose and lung congestion, and fever with anhidrosis, which is the inability to sweat normally (Lee, et al. 2000).

Ephedra contains alkaloids including ephedrine, pseudoephedrine (isoephedrine), norpseudoephedrine (cathine), norephedrine, methylephedrine, methylpseudoephedrine, tannins, and other constituents (Sigma-Aldrich 2015).  The alkaloids ephedrine and pseudoephedrine are found in the leaves and stems. They are structurally similar to amphetamines. They elicit effects of central nervous system stimulation, bronchodilation, hypertension, and chronotropic/inotropic effects. These alkaloids are not present in the New World species of Ephedra (Caveney et al. 2001). Tannins in Ephedra have astringent properties and have long been used in topical preparations to treat oozing and weeping of skin lesions, and also have been thought to possess some renal protective agents (Dashtdar, et al. 2013).  In a GC-MS study, ephedrine was reported to be the major alkaloid in ephedra.  It is found primarily in the stem, with less in the roots (Lv et al. 2015). Another analysis revealed that Ephedra from Mongolia contained high amounts of five ephedrine alkaloids, allowing it to be an effective supplemental resource (Kitani et al. 2009). The Ephedra population in southwestern Mongolia showed a high likelihood of having hybrid origins. The plants in this area were hard to identify based on external features, such as having irregularly curved or twisted stems and several species were involved. They were collected and treated as Ephedra sp., undergoing molecular analysis. It was determined that these were species of Ephedra but showed a higher rate of nucleotide variation (Kitani, et al. 2011).

A study by Song et al. (2012) conducted on mice fed high-fat diets revealed that Ephedra sinica effectively reduced weight gain and epididymal fat accumulation, improved glucose tolerance, decreased triglycerides and increased high-density lipoprotein cholesterol compared to control groups. Those results suggested that Ephedra sinica may reduce obesity and hyperglycemia and could possibly be used clinically to treat glucose intolerance obesity (Song, et al. 2012).

Li et al. (2009) found that in rats with induced spinal cord injuries, administering Ephedra sinica improved motor function improvement after seven days in comparison to a control group given water. Li, et al. (2009) concluded that Ephedra sinica may help reduce inflammation and improve motor function by inhibiting the complement system that causes the inflammation to occur initially.

Conversely, Ephedra has well established adverse effects.  In the 1990s and early 2000s, Ephedra was marketed for weight-loss and energy-enhancement in the United States and were unregulated with no standardization of strength or purity.  There were no proven benefits except for minimal, short-term weight loss. Research has linked Ephedra use with hypertension, cardiac dysrhythmias, myocardial infarction, seizure, stroke, and sudden death (Zell-Kanter, et al. 2015). Dietary supplements containing Ma Huang (Ephedra alkaloids) and guarana (caffeine) were widely marketed despite a lack of research. The quantities of 35 commercial supplements were compared with product labels. The results revealed the total Ephedra alkaloid content ranged from 5.97mg to 29.3mg per serving. Of these supplements, 31% contained greater than 110% of the total Ephedra alkaloids listed on the labeling and 6% contained less than 90% of the listed amount (Haller et.al. 2004).

A study was conducted on the relative toxicity of extracts from Ephedra species under different conditions. Ephedra contains 0.5-2.5% total alkaloids by weight, and ephedrine makes up 30 to 90%. Using high performance liquid chromatography (HPLC), it was determined that there were other chemicals present along with ephedrine, which suggested that there were other unaccounted for toxins present in the extract preparations (Lee, et al. 2000).

In April 2004, the FDA banned the sale of Ephedra-containing products after receiving increasing evidence and more than 18,000 adverse-event reports. The number of calls related to Ephedra use to poison control centers peaked at 10,326 in 2002 and declined to 180 by the end of 2013. There is a direct relation between the ban of Ephedra sales and a decline of adverse-event reports by almost 98% since 2002, proving that the FDA ban on sales was very effective (Zell-Kanter, et al. 2015).

In conclusion, Ephedra does have potential uses, as a stimulant, for weight loss, to treat asthma, among others. However, it does yield adverse reactions, such as hypertension, cardiac dysrhythmias, myocardial infarction, seizure, stroke, and sudden death. Ephedra is an effective plant, but is dangerous without regulation and standardization of dosage. Until there is a standard and the product is regulated when it is sold, it seems that the ban is the best solution to prevent any future issues similar to the ones that occurred in the past.


LITERATURE CITED


Bell, A., & S. Bachman. 2011.  Ephedra sinica. The IUCN Red List of Threatened Species. 2011. http://www.iucnredlist.org/details/201702/0

Caveney, S., D.A. Charlet, H. Freitag, M. Maier-Stolte, & A.N. Starratt.  2001.  New observations on the secondary chemistry of world Ephedra (Ephedraceae). Am J Bot 88.7: 1199-208.

Dashtdar, M., M.R. Dashtdar, B. Dashtdar, M.K. Shirazi, & S.A. Khan. 2013. In vitro, anti-bacterial activities of aqueous extracts of Acacia catechu (L.F.) Willd, Castanea sativa, Ephedra sinica Stapf. and Shilajita mumiyo against Gram positive and Gram negative bacteria. J Pharmacopuncture 16.2: 15-22.

FNA. n.d.  Ephedra Linnaeus. Flora of North America. Volume 2. http://www.efloras.org/florataxon.aspx?flora_id=1&taxon_id=111784.  Accessed 22 June 2015.

Haller, C. A., M. Duan, N.L. Benowitz, & P. Jacob 3rd.  2004.  Concentrations of ephedra alkaloids and caffeine in commercial dietary supplements. J Anal Toxicol 28.3: 145-51.

ITIS 2010.  Ephedra L. Taxonomic Serial 83496.  Integrated Taxonomic Information System. http://www.itis.gov/servlet/SingleRpt/SingleRpt?search_topic=TSN&search_value=183496.   Accessed 22 June 2015.

Kitani, Y., S. Zhu, T. Omote, K. Tanaka, J. Batkhuu, C. Sanchir, H. Fushimi, M. Mikage, & K. Komatsu.  2009.  Molecular analysis and chemical evaluation of ephedra plants in Mongolia. Biol Pharm Bull 32.7: 1235-43.

Kitani, Y., S. Zhu, J. Batkhuu, C. Sanchir, & K. Komatsu.  2011.  Genetic diversity of Ephedra plants in Mongolia inferred from internal transcribed spacer sequence of nuclear ribosomal DNA. Biol Pharm Bull 34.5: 717-26.

Lee, M.K., B.W. Cheng, C.T. Che, & D.P. Hsieh. 2000.  Cytotoxicity assessment of mahuang (Ephedra) under different conditions of preparation. Toxicol Sci 56.2: 424-30.

Li, L., J. Li, Y. Zhu, & G. Fan. 2009. Ephedra sinica inhibits complement activation and improves the motor functions after spinal cord injury in rats.  Brain Res Bull. 16.78(4-5): 261-6.

Lv, M., J. Sun, M. Wang, W. Huang, H. Fan, F. Xu, & Z. Zhang.  2015.  GC-MS based metabolics study of stems and roots of Ephedra sinicaJ Pharm Biomed Anal. 114: 49-52.

Meyer, S.E.  N.D.  Ephedra L. United States Forest Service.  http://www.fs.fed.us/rm/pubs_other/wo_AgricHandbook727/wo_AgricHandbook727_492_494.pdf.   Accessed 22 June 2015.

Sigma-Aldrich 2015.  Ephedra (Ephedra sinica).  Sigma-Aldrich.  http://www.sigmaaldrich.com/life-science/nutrition-research/learning-center/plant-profiler/ephedra-sinica.html. Accessed 22 June 2015.

Song, M.K., J.Y. Um, H.J. Jang, & B.C. Lee. 2012.  Beneficial effect of dietary Ephedra sinica on obesity and glucose intolerance in high-fat diet-fed mice. Exp Ther Med 3.4: 707-12.

Zell-Kanter, M, M.A. Quigley, & J.B. Leikin. 2015. Reduction of Ephedra poisonings after FDA ban. The New England Journal of Medicine: 372:2172-74.


This paper was developed as part of the BIO 368 - Medical Botany course offered at Wilkes University during the summer of 2015. Course instructor was Kenneth M. Klemow, Ph.D. (kenneth.klemow@wilkes.edu). The information contained herein is based on published sources, and is made available for academic purposes only. No warrantees, expressed or implied, are made about the medical usefulness or dangers associated with the plant species in question.

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This page posted and maintained by Kenneth M. Klemow, Ph.D., Biology Department, Wilkes University, Wilkes-Barre, PA 18766. (570) 408-4758, kenneth.klemow@wilkes.edu.